Start Date

2019

Description

  • Antibiotic resistance comes from bacterial mutations in the antibiotic binding site (which prevents antibiotic binding)
  • These new antibiotics target both DNA Gyrase and DNA Topoisomerase IV (fluoroquinolones)
  • This improves efficacy because if one binding site is mutated, the other is still available
  • This makes it harder for bacteria to form resistance against the drug

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Jan 1st, 12:00 AM

Determining the Effectiveness of Drugs using EC50 and MIC Assays

  • Antibiotic resistance comes from bacterial mutations in the antibiotic binding site (which prevents antibiotic binding)
  • These new antibiotics target both DNA Gyrase and DNA Topoisomerase IV (fluoroquinolones)
  • This improves efficacy because if one binding site is mutated, the other is still available
  • This makes it harder for bacteria to form resistance against the drug